Issue |
E3S Web of Conf.
Volume 553, 2024
2024 International Conference on Ecological Protection and Environmental Chemistry (EPEC 2024)
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Article Number | 05030 | |
Number of page(s) | 4 | |
Section | Medical Treatments and Therapies | |
DOI | https://doi.org/10.1051/e3sconf/202455305030 | |
Published online | 24 July 2024 |
Drugs and clinical prospects for the treatment of atherosclerosis
1 Sports and Health College, Shenyang Sport University, 110115 Shenyang, China
2 Department of microbiology, University of Rochester, 14627 Rochester, United States
3 The college of Life Sciences, Sichuan University, 610065 Chengdu, China
4 China-Canada International Department, Hefei No. 1 School, 230000 Hefei, China
* Corresponding author: is_xumy@stu.scu.edu.cn
Atherosclerosis is a disease that occurs in blood vessels and is also responsible for numerous diseases, including coronary heart disease and cerebral infarction. It can affect the function of many organs of the human body, cause great damage to human health, and the incidence is high in the middle-aged and elderly people. The pathogenesis of atherosclerosis is very complex, which is the result of inflammation caused by lipid deposition on the blood vessel wall. At present, aspirin is the most commonly used drug in clinic. It has strong anti-inflammatory and antithrombotic effects. In addition, high density lipoprotein, or HDL, can transport cholesterol in the blood vessels, and it does exert some influence on the treatment of atherosclerosis, but the exact therapeutic pathway is still the subject of ongoing research. This paper illustrates the pathogenesis of atherosclerosis, and introduces the mechanism and feasibility of two therapeutic modalities, aspirin and HDL, and gives a reasonable future research direction.
© The Authors, published by EDP Sciences, 2024
This is an Open Access article distributed under the terms of the Creative Commons Attribution License 4.0, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.
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