Analysis of the genetic predisposition to develop a Myocardial Infarction in a sample of Moroccan patients

Fatima Ezzahra Anaibar; Sana Essenhagi; Said Nassor Abeid; Hamid Farhane; Majida Motrane; Rachida Habbal; Norredine Ghalim; Nourdin Harich

doi:10.1051/e3sconf/202131901013

All issues

Volume 319 (2021)

E3S Web Conf., 319 (2021) 01013

References

Open Access

Issue		E3S Web Conf. Volume 319, 2021 International Congress on Health Vigilance (VIGISAN 2021)


Article Number		01013
Number of page(s)		6
DOI		https://doi.org/10.1051/e3sconf/202131901013
Published online		09 November 2021

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M.A. Batzer, et al., Genetic variation of recent Alu insertions in human population, in Journal of molecular evolution, 42(1): p. 22–29, (1996) [Google Scholar]
D. Swofford, and R. Selander, BIOSYS-1: a computer program for the analysis of allelic variation, in population genetics and biochemical systematics, (1989) [Google Scholar]
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M. Ganesan, et al., The relationship of ACE and CETP gene polymorphisms with cardiovascular disease in a cohort of Asian Indian patients with and those without type 2 diabetes, in Journal of Diabetes and its Complications, 25(5): p. 303–308, (2011) [Google Scholar]
S. Mehri, et al., Angiotensinogen gene polymorphism in acute myocardial infarction patients, in Journal of the Renin-Angiotensin-Aldosterone System, 12(1): p. 42–47, (2011) [Google Scholar]
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[1] H. Zhang, et al., Hirudin protects against isoproternol-induced myocardial infraction by alleviating oxidative via an Nrf2 dependent manner, in International journal of biological macromolecules, 162: p. 425–435, (2020) [Google Scholar]

[2] J.Daccache, et al, Les Canadiens partiellement Français sont susceptibles à une augmentation des facteurs de risque cardiovasculaire: une étude de cohorte rétrospective basée sur la population, in Annales de Cardiologie et d’Angéiologie, Elsevier, (2020) [Google Scholar]

[3] P. Libby, What have we learned about the biology of atherosclerosis? The role of inflammation, in The American journal of cardiology, 88(7): p. 3–6, (2001) [Google Scholar]

[4] M.A. Batzer, and P.L. Deininger, Alu repeats and human genomic diversity, in Nature reviews genetics, 3(5): p. 370–379, (2002) [Google Scholar]

[5] W.S.Watkins, et al., Genetic variation among world populations: inferences from 100 Alu insertion polymorphisms, in Genome research, 13(7): p. 1607–1618, (2003) [Google Scholar]

[6] S.N. Abeid, et al., Human population genetics of Comoros islanders: Alu polymorphisms and the peopling of the three major islands, in Gene Reports, 22: p. 100927, (2021) [Google Scholar]

[7] L. Tiret, et al., Evidence, from combined segregation and linkage analysis, that a variant of the angiotensin I-converting enzyme (ACE) gene controls plasma ACE levels, in American journal of human genetics, 51(1): p. 197, (1992) [Google Scholar]

[8] M.A. Batzer, et al., Genetic variation of recent Alu insertions in human population, in Journal of molecular evolution, 42(1): p. 22–29, (1996) [Google Scholar]

[9] D. Swofford, and R. Selander, BIOSYS-1: a computer program for the analysis of allelic variation, in population genetics and biochemical systematics, (1989) [Google Scholar]

[10] Spss, I., IBM SPSS Statistics for Windows, version 25, in Armonk, NY: IBM SPSS Corp,[Google Scholar],( 2017) [Google Scholar]

[11] P. Frossard, et al., Genetic markers for coronary atherosclerosis in the human apolipoprotein AI-CIII-AIV gene complex, in Am J Hum Genet, 39(Suppl 589), (1986) [Google Scholar]

[12] D. Lim, et al., Deletion of an Alu sequence in the 5′ of the apolipoprotein AI gene associated with decreased serum HDL-cholesterol levels, in Am J Hum Genet, 39: p. 621, (1986) [Google Scholar]

[13] J.L. Breslow, Transgenic mouse models of lipoprotein metabolism and atherosclerosis, in Proceedings of the National Academy of Sciences, 90(18): p. 8314–8318, (1993) [Google Scholar]

[14] A. Falchi, et al., Prevalence of genetic risk factors for coronary artery disease in Corsica island (France), in Experimental and molecular pathology, 79(3): p. 210–213, ( 2005) [Google Scholar]

[15] J.G. Van der Bom, et al., Tissue plasminogen activator and risk of myocardial infarction: the Rotterdam Study, in Circulation, 95(12): p. 2623–2627, (1997) [Google Scholar]

[16] C. Jern, et al., Gene polymorphism of t-PA is associated with forearm vascular release rate of t-PA, in Arteriosclerosis, thrombosis, and vascular biology, 19(2): p. 454–459, (1999) [Google Scholar]

[17] B. Agerholm-Larsen, B.G. Nordestgaard, and A. Tybjærg-Hansen, ACE gene polymorphism in cardiovascular disease: meta-analyses of small and large studies in whites, in Arteriosclerosis, thrombosis, and vascular biology, 20(2): p. 484–492, (2000) [Google Scholar]

[18] Y.Chen, et al., Angiotensin-converting enzyme insertion/deletion polymorphism and risk of myocardial infarction in an updated meta-analysis based on 34 993 participants, in Gene,522(2): p. 196–205, (2013) [Google Scholar]

[19] O. Ozturk, et al., Influence of angiotensin-converting enzyme I/D gene polymorphism on the right ventricular myocardial performance index in patients with a first acute anterior myocardial infarction, in Circulation Journal, 69(2): p. 211–215, (2005) [Google Scholar]

[20] M. Ganesan, et al., The relationship of ACE and CETP gene polymorphisms with cardiovascular disease in a cohort of Asian Indian patients with and those without type 2 diabetes, in Journal of Diabetes and its Complications, 25(5): p. 303–308, (2011) [Google Scholar]

[21] S. Mehri, et al., Angiotensinogen gene polymorphism in acute myocardial infarction patients, in Journal of the Renin-Angiotensin-Aldosterone System, 12(1): p. 42–47, (2011) [Google Scholar]

[22] W.C. Hooper, et al., Relationship of venous thromboembolism and myocardial infarction with the renin-angiotensin system in African-Americans, in American journal of hematology, 70(1): p. 1–8, (2002) [Google Scholar]